Bruxism: its multiple causes and its effects on dental implants – an updated review
Identifieur interne : 007003 ( Main/Exploration ); précédent : 007002; suivant : 007004Bruxism: its multiple causes and its effects on dental implants – an updated review
Auteurs : F. Lobbezoo [Pays-Bas] ; J. Van Der Zaag [Pays-Bas] ; M. Naeije [Pays-Bas]Source :
- Journal of Oral Rehabilitation [ 0305-182X ] ; 2006-04.
English descriptors
- KwdEn :
- Aetiology, Blackwell publishing, Bruxers, Bruxism, Case report, Case reports, Case series, Causal relationship, Central dopaminergic system, Cerebral palsy, Dent, Dental implants, Future research, Future studies, Implant, Implant failure, Kato, Lavigne, Lobbezoo, Main focus, Naeije, Occlusal, Occlusal factors, Oral rehabil, Oral rehabilitation, Orofac pain, Pathophysiological, Possible relevance, Possible role, Practical guidelines, Previous reviews, Psychosocial, Psychosocial factors, Search strategy, Severe bruxism, Temporomandibular disorders.
- Teeft :
- Aetiology, Blackwell publishing, Bruxers, Bruxism, Case report, Case reports, Case series, Causal relationship, Central dopaminergic system, Cerebral palsy, Dent, Dental implants, Future research, Future studies, Implant, Implant failure, Kato, Lavigne, Lobbezoo, Main focus, Naeije, Occlusal, Occlusal factors, Oral rehabil, Oral rehabilitation, Orofac pain, Pathophysiological, Possible relevance, Possible role, Practical guidelines, Previous reviews, Psychosocial, Psychosocial factors, Search strategy, Severe bruxism, Temporomandibular disorders.
Abstract
summary There is a growing interest in bruxism, as evidenced by the rapidly increasing number of papers about this subject during the past 5 years. The aim of the present review was to provide an update of two previous reviews from our department (one about the aetiology of bruxism and the other about the possible role of this movement disorder in the failure of dental implants) and to describe the details of the literature search strategies used, thus enabling the readers to judge the completeness of the review. Most studies that were published about the etiology during the past 5 years corroborate the previously drawn conclusions. Similarly, the update of the review about the possible causal relationship between bruxism and implant failure reveals no new points of view. Thus, there is no reason to assume otherwise than that bruxism is mainly regulated centrally, not peripherally, and that there is still insufficient evidence to support or refute a causal relationship between bruxism and implant failure. This illustrates that there is a vast need for well‐designed studies to study both the aetiology of bruxism and its purported relationship with implant failure.
Url:
DOI: 10.1111/j.1365-2842.2006.01609.x
Affiliations:
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- to stream Main, to step Curation: 007003
Le document en format XML
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<term>Case reports</term>
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<term>Central dopaminergic system</term>
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<term>Implant failure</term>
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<term>Main focus</term>
<term>Naeije</term>
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<term>Occlusal factors</term>
<term>Oral rehabil</term>
<term>Oral rehabilitation</term>
<term>Orofac pain</term>
<term>Pathophysiological</term>
<term>Possible relevance</term>
<term>Possible role</term>
<term>Practical guidelines</term>
<term>Previous reviews</term>
<term>Psychosocial</term>
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<front><div type="abstract">summary There is a growing interest in bruxism, as evidenced by the rapidly increasing number of papers about this subject during the past 5 years. The aim of the present review was to provide an update of two previous reviews from our department (one about the aetiology of bruxism and the other about the possible role of this movement disorder in the failure of dental implants) and to describe the details of the literature search strategies used, thus enabling the readers to judge the completeness of the review. Most studies that were published about the etiology during the past 5 years corroborate the previously drawn conclusions. Similarly, the update of the review about the possible causal relationship between bruxism and implant failure reveals no new points of view. Thus, there is no reason to assume otherwise than that bruxism is mainly regulated centrally, not peripherally, and that there is still insufficient evidence to support or refute a causal relationship between bruxism and implant failure. This illustrates that there is a vast need for well‐designed studies to study both the aetiology of bruxism and its purported relationship with implant failure.</div>
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